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Registro Completo |
Biblioteca(s): |
Biblioteca Rui Tendinha. |
Data corrente: |
14/02/2017 |
Data da última atualização: |
14/02/2017 |
Autoria: |
SILVA, E. de O. e. |
Título: |
Os bancos de dados e o direito a privacidade. |
Ano de publicação: |
1980 |
Fonte/Imprenta: |
Brasília, DF : EMBRAPA-DMQ, 1980. |
Páginas: |
15 p. |
Série: |
(DMQ/B/17). |
Idioma: |
Português |
Conteúdo: |
A segurança de informação armazenado em computadores. Controle de acesso e uso de informacões em bancos de dados. Bancos de Dados: uma ameaça ao direito ao singilo e a privacidade? Coleta, armazenamento e disseminação de informações armazenadas em computadores : aspectos éticos e legais. |
Palavras-Chave: |
Armazenagem; Armazenamento; Armazenamento de informacao; Banco; Banco de dado; Banco de dados; Banco de dados - Seguranca; Bancos de dados; Computador; Computer; Computer applicatiions; Dado; Dados; Data bank; Data bases; Databases; Diffusion of information; Disseminacao de informacao; Informação; Informática; Information; Information storage; Privacidade; Security; Seguranca; Sewrity; Storage. |
Categoria do assunto: |
-- |
Marc: |
LEADER 01440nam a2200457 a 4500 001 1014267 005 2017-02-14 008 1980 bl uuuu u0uu1 u #d 100 1 $aSILVA, E. de O. e. 245 $aOs bancos de dados e o direito a privacidade. 260 $aBrasília, DF : EMBRAPA-DMQ$c1980 300 $a15 p. 490 $a(DMQ/B/17). 520 $aA segurança de informação armazenado em computadores. Controle de acesso e uso de informacões em bancos de dados. Bancos de Dados: uma ameaça ao direito ao singilo e a privacidade? Coleta, armazenamento e disseminação de informações armazenadas em computadores : aspectos éticos e legais. 653 $aArmazenagem 653 $aArmazenamento 653 $aArmazenamento de informacao 653 $aBanco 653 $aBanco de dado 653 $aBanco de dados 653 $aBanco de dados - Seguranca 653 $aBancos de dados 653 $aComputador 653 $aComputer 653 $aComputer applicatiions 653 $aDado 653 $aDados 653 $aData bank 653 $aData bases 653 $aDatabases 653 $aDiffusion of information 653 $aDisseminacao de informacao 653 $aInformação 653 $aInformática 653 $aInformation 653 $aInformation storage 653 $aPrivacidade 653 $aSecurity 653 $aSeguranca 653 $aSewrity 653 $aStorage
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Registro original: |
Biblioteca Rui Tendinha (BRT) |
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Registro Completo |
Biblioteca(s): |
Biblioteca Rui Tendinha. |
Data corrente: |
19/01/2021 |
Data da última atualização: |
19/01/2021 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
A - 1 |
Autoria: |
SÁ ANTUNES, T. F.; MAURASTONI, M. L.; MADROÑERO, J.; FUENTES, G.; SANTAMARÍA, J. M.; VENTURA, J. A.; ABREU, E. F.; FERNANDES, A. R.; FERNANDES, P. M. B. |
Afiliação: |
Tathiana F. Sá Antunes, UFES; Marlonni Maurastoni L., UFES; Johana Madroñero, UFES/UNIVERSIDAD EL BOSQUE; Gabriela Fuentes, Centro de Investigación Científica de Yucatán; Jorge M. Santamaría, Centro de Investigación Científica de Yucatán; Jose Aires Ventura, Incaper; Emauel F. Abreu, Embrapa Recursos Genéticos e Biotecnologia; Alberto R. Fernandes, UFES; Patricia M. B. Fernandes, UFES. |
Título: |
Battle of three: the curious case of papaya sticky disease. |
Ano de publicação: |
2020 |
Fonte/Imprenta: |
Plant Disease, v. 104, n. 11, p. 2754-2763, 2020. |
Idioma: |
Português |
Conteúdo: |
Among the most serious problems in papaya production are the viruses associated with papaya ringspot and papaya sticky disease (PSD). PSD concerns producers worldwide because its symptoms are extremely aggressive and appear only after flowering. As no resistant cultivar is available, several disease management strategies have been used in affected countries, such as the use of healthy seeds, exclusion of the pathogen, and roguing. In the 1990s, a dsRNA virus, papaya meleira virus (PMeV), was identified in Brazil as the causal agent of PSD. However, in 2016 a second virus, papaya meleira virus 2 (PMeV2), with an ssRNA genome, was also identified in PSD plants. Only PMeV is detected in asymptomatic plants, whereas all symptomatic plants contain both viral RNAs separately packaged in particles formed by the PMeV capsid protein. PSD also affects papaya plants in Mexico, Ecuador, and Australia. PMeV2-like viruses have been identified in the affected plants, but the partner virus(es) in these countries are still unknown. In Brazil, PMeV and PMeV2 reside in laticifers that promote spontaneous latex exudation, resulting in the affected papaya fruit?s sticky appearance. Genes modulated in plants affected by PSD include those involved in reactive oxygen species and salicylic acid signaling, proteasomal degradation, and photosynthesis, which are key plant defenses against PMeV complex infection. However, the complete activation of the defense response is impaired by the expression of negative effectors modulated by the virus. This review presents a summary of the current knowledge of the Carica papaya-PMeV complex interaction and management strategies. MenosAmong the most serious problems in papaya production are the viruses associated with papaya ringspot and papaya sticky disease (PSD). PSD concerns producers worldwide because its symptoms are extremely aggressive and appear only after flowering. As no resistant cultivar is available, several disease management strategies have been used in affected countries, such as the use of healthy seeds, exclusion of the pathogen, and roguing. In the 1990s, a dsRNA virus, papaya meleira virus (PMeV), was identified in Brazil as the causal agent of PSD. However, in 2016 a second virus, papaya meleira virus 2 (PMeV2), with an ssRNA genome, was also identified in PSD plants. Only PMeV is detected in asymptomatic plants, whereas all symptomatic plants contain both viral RNAs separately packaged in particles formed by the PMeV capsid protein. PSD also affects papaya plants in Mexico, Ecuador, and Australia. PMeV2-like viruses have been identified in the affected plants, but the partner virus(es) in these countries are still unknown. In Brazil, PMeV and PMeV2 reside in laticifers that promote spontaneous latex exudation, resulting in the affected papaya fruit?s sticky appearance. Genes modulated in plants affected by PSD include those involved in reactive oxygen species and salicylic acid signaling, proteasomal degradation, and photosynthesis, which are key plant defenses against PMeV complex infection. However, the complete activation of the defense response is impaired by the expression of n... Mostrar Tudo |
Palavras-Chave: |
Meleira. |
Thesagro: |
Carica Papaya; Doença; Mamão; Praga. |
Categoria do assunto: |
H Saúde e Patologia |
URL: |
https://biblioteca.incaper.es.gov.br/digital/bitstream/123456789/4168/1/Battle-of-Three-Papaya-Sticky-Disease-2020.pdf
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Marc: |
LEADER 02409naa a2200277 a 4500 001 1023094 005 2021-01-19 008 2020 bl uuuu u00u1 u #d 100 1 $aSÁ ANTUNES, T. F. 245 $aBattle of three$bthe curious case of papaya sticky disease.$h[electronic resource] 260 $c2020 520 $aAmong the most serious problems in papaya production are the viruses associated with papaya ringspot and papaya sticky disease (PSD). PSD concerns producers worldwide because its symptoms are extremely aggressive and appear only after flowering. As no resistant cultivar is available, several disease management strategies have been used in affected countries, such as the use of healthy seeds, exclusion of the pathogen, and roguing. In the 1990s, a dsRNA virus, papaya meleira virus (PMeV), was identified in Brazil as the causal agent of PSD. However, in 2016 a second virus, papaya meleira virus 2 (PMeV2), with an ssRNA genome, was also identified in PSD plants. Only PMeV is detected in asymptomatic plants, whereas all symptomatic plants contain both viral RNAs separately packaged in particles formed by the PMeV capsid protein. PSD also affects papaya plants in Mexico, Ecuador, and Australia. PMeV2-like viruses have been identified in the affected plants, but the partner virus(es) in these countries are still unknown. In Brazil, PMeV and PMeV2 reside in laticifers that promote spontaneous latex exudation, resulting in the affected papaya fruit?s sticky appearance. Genes modulated in plants affected by PSD include those involved in reactive oxygen species and salicylic acid signaling, proteasomal degradation, and photosynthesis, which are key plant defenses against PMeV complex infection. However, the complete activation of the defense response is impaired by the expression of negative effectors modulated by the virus. This review presents a summary of the current knowledge of the Carica papaya-PMeV complex interaction and management strategies. 650 $aCarica Papaya 650 $aDoença 650 $aMamão 650 $aPraga 653 $aMeleira 700 1 $aMAURASTONI, M. L. 700 1 $aMADROÑERO, J. 700 1 $aFUENTES, G. 700 1 $aSANTAMARÍA, J. M. 700 1 $aVENTURA, J. A. 700 1 $aABREU, E. F. 700 1 $aFERNANDES, A. R. 700 1 $aFERNANDES, P. M. B. 773 $tPlant Disease$gv. 104, n. 11, p. 2754-2763, 2020.
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